Skip to content
The Longevity Archive
All compounds
Muscle & PerformanceResearch strength: PreliminaryEvidence: Preliminary

ACE-031

ACE-031 (soluble activin receptor type IIB)

Also known as: Ramatercept

A myostatin/activin decoy studied for muscular dystrophy whose trials were halted over vascular safety signals.

Overview

ACE-031 is a soluble form of the activin receptor type IIB that acts as a decoy, trapping myostatin and related factors to promote muscle growth. It was studied for Duchenne muscular dystrophy, but trials were halted in 2011 after signals including nosebleeds and small vessel changes. It is not approved.

Mechanism of action

Binds and neutralizes myostatin and activin before they can signal, releasing their inhibition of muscle growth.

Key studies & citations

  • Human2013

    A single ascending-dose study of muscle regulator ACE-031 in healthy volunteers

    One dose in 48 postmenopausal women: half-life 10 to 15 days; at 3 mg/kg total lean mass rose 3.3% and thigh muscle volume 5.1% by day 29.

    Muscle and Nerve
  • Human2017

    Myostatin inhibitor ACE-031 treatment of ambulatory boys with Duchenne muscular dystrophy: results of a randomized, placebo-controlled clinical trial

    The trial was stopped after the second dose level because of epistaxis and telangiectasias; only non-significant trends toward preserved six-minute walk and higher lean mass were seen.

    Muscle and Nerve

Frequently asked questions

Why was ACE-031 discontinued?

Its trials were halted after non-muscle safety signals, including nosebleeds and small blood vessel changes, outweighed the muscle benefits.

Related compounds